Dihydromyricetin alleviates carbon tetrachloride-induced acute liver injury via JNK-dependent mechanism in mice. [electronic resource]
Producer: 20160411Description: 5473-81 p. digitalISSN:- 2219-2840
- Animals
- Anti-Inflammatory Agents -- pharmacology
- Biomarkers -- blood
- Carbon Tetrachloride
- Caspase Inhibitors -- pharmacology
- Cell Proliferation -- drug effects
- Chemical and Drug Induced Liver Injury -- blood
- Cytochromes c -- metabolism
- Disease Models, Animal
- Flavonols -- pharmacology
- Inflammation Mediators -- blood
- JNK Mitogen-Activated Protein Kinases -- antagonists & inhibitors
- Liver -- drug effects
- Liver Failure, Acute -- blood
- Liver Regeneration -- drug effects
- Male
- Mice, Inbred C57BL
- Mitochondria, Liver -- drug effects
- Protein Kinase Inhibitors -- pharmacology
- Signal Transduction -- drug effects
- Time Factors
- Tumor Necrosis Factor-alpha -- blood
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.