Estrogen and ERα enhanced β-catenin degradation and suppressed its downstream target genes to block the metastatic function of HA22T hepatocellular carcinoma cells via modulating GSK-3β and β-TrCP expression. [electronic resource]
Producer: 20170301Description: 519-529 p. digitalISSN:- 1522-7278
- Carcinoma, Hepatocellular -- metabolism
- Cell Line, Tumor
- Cell Movement
- Down-Regulation
- Estrogen Receptor alpha -- genetics
- Estrogen Receptor beta -- genetics
- Glycogen Synthase Kinase 3 beta -- genetics
- Humans
- Immunohistochemistry
- Immunoprecipitation
- Liver Neoplasms -- metabolism
- Microscopy, Fluorescence
- beta Catenin -- metabolism
- beta-Transducin Repeat-Containing Proteins -- genetics
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Publication Type: Journal Article
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