APA

Raimondo A., Chakera A. J., Thomsen S. K., Colclough K., Barrett A., De Franco E., Chatelas A., Demirbilek H., Akcay T., Alawneh H., Flanagan S. E., Van De Bunt M., Hattersley A. T., Gloyn A. L. & Ellard S. (20150709). Phenotypic severity of homozygous GCK mutations causing neonatal or childhood-onset diabetes is primarily mediated through effects on protein stability. : Human molecular genetics.

Chicago

Raimondo Anne, Chakera Ali J, Thomsen Soren K, Colclough Kevin, Barrett Amy, De Franco Elisa, Chatelas Alisson, Demirbilek Huseyin, Akcay Teoman, Alawneh Hussein, Flanagan Sarah E, Van De Bunt Martijn, Hattersley Andrew T, Gloyn Anna L and Ellard Sian. 20150709. Phenotypic severity of homozygous GCK mutations causing neonatal or childhood-onset diabetes is primarily mediated through effects on protein stability. : Human molecular genetics.

Harvard

Raimondo A., Chakera A. J., Thomsen S. K., Colclough K., Barrett A., De Franco E., Chatelas A., Demirbilek H., Akcay T., Alawneh H., Flanagan S. E., Van De Bunt M., Hattersley A. T., Gloyn A. L. and Ellard S. (20150709). Phenotypic severity of homozygous GCK mutations causing neonatal or childhood-onset diabetes is primarily mediated through effects on protein stability. : Human molecular genetics.

MLA

Raimondo Anne, Chakera Ali J, Thomsen Soren K, Colclough Kevin, Barrett Amy, De Franco Elisa, Chatelas Alisson, Demirbilek Huseyin, Akcay Teoman, Alawneh Hussein, Flanagan Sarah E, Van De Bunt Martijn, Hattersley Andrew T, Gloyn Anna L and Ellard Sian. Phenotypic severity of homozygous GCK mutations causing neonatal or childhood-onset diabetes is primarily mediated through effects on protein stability. : Human molecular genetics. 20150709.