Neutrophil apoptosis is delayed by trauma patients' plasma via a mechanism involving proinflammatory phospholipids and protein kinase C. [electronic resource]
Producer: 20030312Description: 289-93; discussion 294-5 p. digitalISSN:- 1096-2964
- Adolescent
- Adult
- Aged
- Apoptosis -- drug effects
- Azepines -- pharmacology
- Enzyme Inhibitors -- pharmacology
- Humans
- In Vitro Techniques
- Indoles -- pharmacology
- Injury Severity Score
- Maleimides -- pharmacology
- Middle Aged
- Neutrophils -- drug effects
- Phospholipids -- immunology
- Plasma -- drug effects
- Platelet Activating Factor -- drug effects
- Platelet Aggregation Inhibitors -- pharmacology
- Protein Kinase C -- drug effects
- Signal Transduction -- drug effects
- Time Factors
- Triazoles -- pharmacology
- Wounds and Injuries -- immunology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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