Long-term but not short-term p38 mitogen-activated protein kinase inhibition improves cardiac function and reduces cardiac remodeling post-myocardial infarction. [electronic resource]
Producer: 20080616Description: 741-50 p. digitalISSN:- 1521-0103
- Actins -- metabolism
- Angiotensin II -- pharmacology
- Angiotensin-Converting Enzyme Inhibitors -- pharmacology
- Animals
- Blood Pressure -- drug effects
- Cells, Cultured
- Collagen -- metabolism
- HSP27 Heat-Shock Proteins
- Heat-Shock Proteins -- metabolism
- Imidazoles -- therapeutic use
- Myocardial Infarction -- drug therapy
- Myocytes, Cardiac -- drug effects
- Neoplasm Proteins -- metabolism
- Organ Size -- drug effects
- Protein Kinase Inhibitors -- therapeutic use
- Pyridines -- therapeutic use
- Ramipril -- pharmacology
- Rats
- Rats, Sprague-Dawley
- Tumor Necrosis Factor-alpha -- blood
- Ventricular Dysfunction, Left -- drug therapy
- Ventricular Remodeling -- drug effects
- p38 Mitogen-Activated Protein Kinases -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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