Changes in cortical and striatal neurons predict behavioral and electrophysiological abnormalities in a transgenic murine model of Huntington's disease. [electronic resource]
Producer: 20020111Description: 9112-23 p. digitalISSN:- 1529-2401
- Age of Onset
- Animals
- Behavior, Animal
- Calcium -- metabolism
- Cell Nucleus -- pathology
- Cerebral Cortex -- pathology
- Corpus Callosum -- physiopathology
- Corpus Striatum -- drug effects
- Dendrites -- pathology
- Disease Models, Animal
- Disease Progression
- Electrophysiology
- Excitatory Amino Acid Agonists -- pharmacology
- Heterozygote
- Huntingtin Protein
- Huntington Disease -- pathology
- In Vitro Techniques
- Mice
- Mice, Transgenic
- Nerve Tissue Proteins -- genetics
- Neurons -- drug effects
- Nuclear Proteins -- genetics
- Phenotype
- Receptors, N-Methyl-D-Aspartate -- metabolism
- Trinucleotide Repeat Expansion
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
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