Anti-Fas induces hepatic chemokines and promotes inflammation by an NF-kappa B-independent, caspase-3-dependent pathway. [electronic resource]
Producer: 20020131Description: 49077-82 p. digitalISSN:- 0021-9258
- Animals
- Antibodies, Monoclonal -- administration & dosage
- Apoptosis -- immunology
- Caspase 3
- Caspase Inhibitors
- Caspases -- metabolism
- Chemokine CXCL1
- Chemokine CXCL2
- Chemokines, CXC -- genetics
- Chemotactic Factors -- metabolism
- Enzyme Inhibitors -- pharmacology
- Female
- Gene Expression Regulation
- Growth Substances -- metabolism
- Hepatitis -- pathology
- In Situ Hybridization
- In Situ Nick-End Labeling
- Intercellular Signaling Peptides and Proteins
- Ligands
- Liver -- drug effects
- Mice
- Monokines -- genetics
- NF-kappa B -- metabolism
- Neutrophil Infiltration
- Tissue Extracts -- chemistry
- Transcription Factor AP-1 -- metabolism
- fas Receptor -- immunology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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