Outer inflammatory protein a (OipA) of Helicobacter pylori is regulated by host cell contact and mediates CagA translocation and interleukin-8 response only in the presence of a functional cag pathogenicity island type IV secretion system. [electronic resource]
Producer: 20180706ISSN:- 2049-632X
- Amplified Fragment Length Polymorphism Analysis
- Antigens, Bacterial -- genetics
- Bacterial Adhesion -- physiology
- Bacterial Outer Membrane Proteins -- genetics
- Bacterial Proteins -- genetics
- Cell Line, Tumor
- Genomic Islands -- genetics
- Helicobacter Infections -- microbiology
- Helicobacter pylori -- genetics
- Host-Pathogen Interactions
- Humans
- Interleukin-8 -- immunology
- Protein Transport -- physiology
- RNA, Messenger -- biosynthesis
- Type IV Secretion Systems -- genetics
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Publication Type: Journal Article
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