Overexpression of GILZ in macrophages limits systemic inflammation while increasing bacterial clearance in sepsis in mice. [electronic resource]
Producer: 20200810Description: 589-602 p. digitalISSN:- 1521-4141
- Animals
- Anti-Inflammatory Agents -- metabolism
- Bacterial Load
- Cells, Cultured
- Cytokines -- metabolism
- Disease Models, Animal
- Gene Expression Regulation
- Glucocorticoids -- metabolism
- Humans
- Immunity, Innate
- Immunomodulation
- Inflammation -- metabolism
- Inflammation Mediators -- metabolism
- Macrophages -- immunology
- Mice
- Mice, Transgenic
- Monocytes -- immunology
- Sepsis -- metabolism
- Transcription Factors -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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