Genetic deletion of the bacterial sensor NOD2 improves murine Crohn's disease-like ileitis independent of functional dysbiosis. [electronic resource]
Producer: 20180326Description: 971-982 p. digitalISSN:- 1935-3456
- Animals
- Colitis -- chemically induced
- Crohn Disease -- genetics
- Cytokines -- metabolism
- Dextran Sulfate
- Disease Models, Animal
- Disease Susceptibility
- Dysbiosis
- Feces -- microbiology
- Humans
- Ileitis -- genetics
- Intestinal Mucosa -- pathology
- Mice
- Mice, Knockout
- Mice, Mutant Strains
- Microbiota -- genetics
- Nod2 Signaling Adaptor Protein -- genetics
- RNA, Ribosomal, 16S -- analysis
- Receptors, Pattern Recognition -- genetics
- STAT6 Transcription Factor -- metabolism
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Publication Type: Journal Article
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