Activin signaling in microsatellite stable colon cancers is disrupted by a combination of genetic and epigenetic mechanisms. [electronic resource]
Producer: 20100317Description: e8308 p. digitalISSN:- 1932-6203
- Activin Receptors, Type I -- genetics
- Activin Receptors, Type II -- genetics
- Cell Line, Tumor
- Colonic Neoplasms -- classification
- DNA Methylation -- genetics
- Epigenesis, Genetic
- Gene Expression Regulation, Neoplastic
- Humans
- Loss of Heterozygosity -- genetics
- Microsatellite Instability
- Microsatellite Repeats -- genetics
- Mutation -- genetics
- Promoter Regions, Genetic -- genetics
- Signal Transduction -- genetics
- Smad2 Protein -- genetics
- Transcription, Genetic
- Tumor Burden -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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