Caffeic acid improves locomotor activity and lessens inflammatory burden in a mouse model of rotenone-induced nigral neurodegeneration: Relevance to Parkinson's disease therapy. [electronic resource]
Producer: 20190403Description: 32-41 p. digitalISSN:- 2299-5684
- Animals
- Anti-Inflammatory Agents -- pharmacology
- Antiparkinson Agents -- pharmacology
- Apoptosis -- drug effects
- Behavior, Animal -- drug effects
- CD11b Antigen -- genetics
- Caffeic Acids -- pharmacology
- Cyclooxygenase 2 -- genetics
- Disease Models, Animal
- Dopamine -- metabolism
- Inflammation Mediators -- metabolism
- Locomotion -- drug effects
- Male
- Mice
- Motor Activity -- drug effects
- NF-kappa B -- metabolism
- Neuroprotective Agents -- pharmacology
- Nitric Oxide Synthase Type II -- genetics
- Parkinsonian Disorders -- chemically induced
- Rotenone
- Substantia Nigra -- drug effects
- Tyrosine 3-Monooxygenase -- metabolism
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Publication Type: Journal Article
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