Smad2-dependent protease nexin-1 overexpression differentiates chronic aneurysms from acute dissections of human ascending aorta. [electronic resource]
Producer: 20131112Description: 2222-32 p. digitalISSN:- 1524-4636
- Acute Disease
- Adult
- Aged
- Aged, 80 and over
- Aortic Dissection -- etiology
- Aortic Aneurysm, Thoracic -- etiology
- Aortic Valve -- abnormalities
- Bicuspid Aortic Valve Disease
- Binding Sites
- Biomarkers -- metabolism
- Cells, Cultured
- Chronic Disease
- Female
- Fibrillins
- Genetic Predisposition to Disease
- Genotype
- Heart Valve Diseases -- complications
- Humans
- Male
- Marfan Syndrome -- complications
- Microfilament Proteins -- genetics
- Middle Aged
- Muscle, Smooth, Vascular -- metabolism
- Mutation
- Myocytes, Smooth Muscle -- metabolism
- Phenotype
- Plasminogen Activator Inhibitor 1 -- metabolism
- Promoter Regions, Genetic
- RNA, Messenger -- metabolism
- Risk Factors
- Serpin E2 -- genetics
- Smad2 Protein -- metabolism
- Time Factors
- Transforming Growth Factor beta1 -- metabolism
- Up-Regulation
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
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