The Familial α-Synuclein A53E Mutation Enhances Cell Death in Response to Environmental Toxins Due to a Larger Population of Oligomers. [electronic resource]
Producer: 20190606Description: 5014-5028 p. digitalISSN:- 1520-4995
- Apoptosis -- drug effects
- Cell Line, Tumor
- Dopamine -- toxicity
- Humans
- Kinetics
- Membrane Potential, Mitochondrial
- Mitochondria -- drug effects
- Mutation
- Protein Aggregates -- genetics
- Protein Aggregation, Pathological -- metabolism
- Protein Multimerization
- Reactive Oxygen Species -- metabolism
- Rotenone -- toxicity
- alpha-Synuclein -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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