Juvenile Huntington's Disease Skin Fibroblasts Respond with Elevated Parkin Level and Increased Proteasome Activity as a Potential Mechanism to Counterbalance the Pathological Consequences of Mutant Huntingtin Protein. [electronic resource]
Producer: 20200413ISSN:- 1422-0067
- Cell Proliferation
- Cells, Cultured
- Fibroblasts -- cytology
- GTP Phosphohydrolases -- metabolism
- Glycolysis
- Humans
- Huntingtin Protein -- genetics
- Huntington Disease -- genetics
- Membrane Potential, Mitochondrial -- physiology
- Mitochondria -- metabolism
- Mitochondrial Membrane Transport Proteins -- metabolism
- Mutation
- Neurons -- metabolism
- Peptides -- metabolism
- Proteasome Endopeptidase Complex -- metabolism
- Reactive Oxygen Species -- metabolism
- Skin -- cytology
- Ubiquitin -- metabolism
- Ubiquitin-Protein Ligases -- genetics
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Publication Type: Journal Article
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