Sobel, E S <br/><br/>
Genetic dissection of SLE pathogenesis: adoptive transfer of Sle1 mediates the loss of tolerance by bone marrow-derived B cells.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Feb 1999 
 - 2415-21 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0022-1767 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adoptive Transfer<br/>Animals<br/>Antigens, CD--biosynthesis<br/>Antigens, Differentiation, T-Lymphocyte--biosynthesis<br/>Autoantibodies--biosynthesis<br/>Autoantigens--biosynthesis<br/>B-Lymphocyte Subsets--immunology<br/>B7-2 Antigen<br/>Bone Marrow Cells--immunology<br/>Bone Marrow Transplantation--immunology<br/>CD4-Positive T-Lymphocytes--transplantation<br/>Gene Expression Regulation--immunology<br/>Genetic Markers--immunology<br/>Immune Tolerance--genetics<br/>Immunoglobulin G--biosynthesis<br/>Immunophenotyping<br/>Lectins, C-Type<br/>Lupus Erythematosus, Systemic--etiology<br/>Membrane Glycoproteins--biosynthesis<br/>Mice<br/>Mice, Congenic<br/>Mice, Inbred C57BL<br/>Mice, Inbred NZB<br/>Nucleosomes--immunology<br/>T-Lymphocyte Subsets--metabolism<br/>Transplantation Chimera--genetics