Sementchenko, V I <br/><br/>
ETS2 function is required to maintain the transformed state of human prostate cancer cells.  [electronic resource] 

 -  Oncogene  Dec 1998 
 - 2883-8 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, U.S. Gov't, Non-P.H.S. 
<br/><br/><label>ISSN: </label>0950-9232 
<br/><br/><label>Standard No.: </label>10.1038/sj.onc.1202220  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Agar<br/>Androgens<br/>Blotting, Northern<br/>Cell Division<br/>Cell Transformation, Neoplastic<br/>DNA-Binding Proteins--genetics<br/>Gene Expression Regulation, Neoplastic<br/>Genes, Dominant<br/>Humans<br/>Male<br/>Phenotype<br/>Precipitin Tests<br/>Prostatic Neoplasms--genetics<br/>Proto-Oncogene Protein c-ets-2<br/>Proto-Oncogene Proteins--antagonists & inhibitors<br/>RNA, Antisense--genetics<br/>Repressor Proteins<br/>Sequence Deletion<br/>Trans-Activators--antagonists & inhibitors<br/>Transcription Factors<br/>Transfection<br/>Tumor Cells, Cultured