Gross, A <br/><br/>
Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death.  [electronic resource] 

 -  The Journal of biological chemistry  Jan 1999 
 - 1156-63 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0021-9258 
<br/><br/><label>Standard No.: </label>10.1074/jbc.274.2.1156  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amino Acid Sequence<br/>Antigens, CD--metabolism<br/>Apoptosis<br/>BH3 Interacting Domain Death Agonist Protein<br/>Carrier Proteins--chemistry<br/>Caspases--metabolism<br/>Cycloheximide--pharmacology<br/>Cytochrome c Group--metabolism<br/>Cytosol--metabolism<br/>Hydrolysis<br/>Mitochondria--drug effects<br/>Molecular Sequence Data<br/>Proto-Oncogene Proteins c-bcl-2--metabolism<br/>Receptors, Tumor Necrosis Factor--metabolism<br/>Receptors, Tumor Necrosis Factor, Type I<br/>Tumor Necrosis Factor-alpha--pharmacology<br/>bcl-X Protein<br/>fas Receptor--metabolism