Cleavage of translation initiation factor 4G (eIF4G) during anti-Fas IgM-induced apoptosis does not require signalling through the p38 mitogen-activated protein (MAP) kinase. [electronic resource]
- FEBS letters Oct 1998
- 41-8 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
0014-5793
10.1016/s0014-5793(98)01269-1 doi
Amino Acid Chloromethyl Ketones--pharmacology Apoptosis--drug effects Arsenites--pharmacology Calcium-Calmodulin-Dependent Protein Kinases--metabolism Caspase Inhibitors Cell Survival--drug effects Etoposide--pharmacology Eukaryotic Initiation Factor-4E Eukaryotic Initiation Factor-4G Humans Imidazoles--pharmacology Immunoglobulin M Jurkat Cells Mitogen-Activated Protein Kinases Peptide Initiation Factors--metabolism Phosphorylation Poly(A)-Binding Proteins Protein Biosynthesis--drug effects Pyridines--pharmacology RNA-Binding Proteins--metabolism Signal Transduction Sirolimus--pharmacology fas Receptor--immunology p38 Mitogen-Activated Protein Kinases