Kupershmidt, S <br/><br/>
A K+ channel splice variant common in human heart lacks a C-terminal domain required for expression of rapidly activating delayed rectifier current.  [electronic resource] 

 -  The Journal of biological chemistry  Oct 1998 
 - 27231-5 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0021-9258 
<br/><br/><label>Standard No.: </label>10.1074/jbc.273.42.27231  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alternative Splicing<br/>Cation Transport Proteins<br/>Cloning, Molecular<br/>DNA-Binding Proteins<br/>ERG1 Potassium Channel<br/>Electric Conductivity<br/>Ether-A-Go-Go Potassium Channels<br/>Humans<br/>Long QT Syndrome--metabolism<br/>Molecular Sequence Data<br/>Myocardium--metabolism<br/>Potassium--metabolism<br/>Potassium Channels--genetics<br/>Potassium Channels, Voltage-Gated<br/>Recombinant Proteins--metabolism<br/>Sequence Analysis, DNA<br/>Sequence Deletion<br/>Trans-Activators<br/>Transcriptional Regulator ERG