Bäumer, A T <br/><br/>
Overexpression of the Na(+)-Ca2+ exchanger leads to enhanced inotropic responsiveness to Na(+)-channel agonist without sarcoplasmic reticulum protein changes in transgenic mice.  [electronic resource] 

 -  Biochemical and biophysical research communications  Aug 1998 
 - 786-90 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0006-291X 
<br/><br/><label>Standard No.: </label>10.1006/bbrc.1998.9248  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Azetidines--pharmacology<br/>Calcium-Binding Proteins--metabolism<br/>Calcium-Transporting ATPases--metabolism<br/>Calsequestrin--metabolism<br/>Dogs<br/>Gene Expression<br/>Immunohistochemistry<br/>In Vitro Techniques<br/>Isoproterenol--pharmacology<br/>Mice<br/>Mice, Inbred C3H<br/>Mice, Inbred C57BL<br/>Mice, Transgenic<br/>Myocardial Contraction--drug effects<br/>Proteins--metabolism<br/>Sarcoplasmic Reticulum--metabolism<br/>Sodium Channel Agonists<br/>Sodium-Calcium Exchanger--genetics