Hennebicq-Reig, S <br/><br/>
Permanent exposure of mucin-secreting HT-29 cells to benzyl-N-acetyl-alpha-D-galactosaminide induces abnormal O-glycosylation of mucins and inhibits constitutive and stimulated MUC5AC secretion.  [electronic resource] 

 -  The Biochemical journal  Aug 1998 
 - 283-95 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0264-6021 
<br/><br/><label>Standard No.: </label>10.1042/bj3340283  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acetylgalactosamine--analogs & derivatives<br/>Adenocarcinoma<br/>Benzyl Compounds--pharmacology<br/>Carbohydrate Sequence<br/>Colonic Neoplasms<br/>Galactose--metabolism<br/>Glycosylation<br/>Glycosyltransferases--metabolism<br/>Humans<br/>Kinetics<br/>Microsomes--enzymology<br/>Molecular Sequence Data<br/>Mucin 5AC<br/>Mucins--biosynthesis<br/>Nuclear Magnetic Resonance, Biomolecular<br/>Oligosaccharides--biosynthesis<br/>Polymerase Chain Reaction<br/>Sialic Acids--metabolism<br/>Tumor Cells, Cultured