Anderson, K L <br/><br/>
Myeloid development is selectively disrupted in PU.1 null mice.  [electronic resource] 

 -  Blood  May 1998 
 - 3702-10 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0006-4971 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cell Differentiation--genetics<br/>Cell Division<br/>Cells, Cultured<br/>Colony-Forming Units Assay<br/>Drug Resistance<br/>Granulocyte Colony-Stimulating Factor--pharmacology<br/>Granulocyte-Macrophage Colony-Stimulating Factor--pharmacology<br/>Hematopoiesis--genetics<br/>Hematopoietic Stem Cells--drug effects<br/>Immunologic Deficiency Syndromes--genetics<br/>Leukopenia--genetics<br/>Lymphocyte Subsets--pathology<br/>Macrophage Colony-Stimulating Factor--pharmacology<br/>Macrophages--pathology<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Monocytes--pathology<br/>Neutrophils--pathology<br/>Phenotype<br/>Proto-Oncogene Proteins--deficiency<br/>Receptor, Macrophage Colony-Stimulating Factor--deficiency<br/>Receptors, Granulocyte Colony-Stimulating Factor--deficiency<br/>Receptors, Granulocyte-Macrophage Colony-Stimulating Factor--deficiency<br/>Trans-Activators--deficiency