Ahmed, M <br/><br/>
BCR-ABL and constitutively active erythropoietin receptor (cEpoR) activate distinct mechanisms for growth factor-independence and inhibition of apoptosis in Ba/F3 cell line.  [electronic resource] 

 -  Oncogene  Jan 1998 
 - 489-96 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0950-9232 
<br/><br/><label>Standard No.: </label>10.1038/sj.onc.1201556  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--physiology<br/>Cell Division--drug effects<br/>Cell Line--cytology<br/>Erythropoietin--metabolism<br/>Fusion Proteins, bcr-abl--genetics<br/>Genetic Vectors<br/>Interleukin-3--pharmacology<br/>Mice<br/>Oncogenes--physiology<br/>Proto-Oncogene Proteins--metabolism<br/>Proto-Oncogene Proteins c-bcl-2--metabolism<br/>Receptors, Erythropoietin--physiology<br/>Signal Transduction<br/>Transfection<br/>bcl-2-Associated X Protein