Bamber, B <br/><br/>
Oncostatin M stimulates excessive extracellular matrix accumulation in a transgenic mouse model of connective tissue disease.  [electronic resource] 

 -  Journal of molecular medicine (Berlin, Germany)  Jan 1998 
 - 61-9 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>0946-2716 
<br/><br/><label>Standard No.: </label>10.1007/s001090050191  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Animals, Newborn<br/>Cattle<br/>Connective Tissue Diseases--metabolism<br/>Crosses, Genetic<br/>Cytokines--genetics<br/>Disease Models, Animal<br/>Extracellular Matrix Proteins--genetics<br/>Fibrosis<br/>Gene Expression Regulation<br/>Growth Substances--genetics<br/>Interleukin-6--genetics<br/>Islets of Langerhans--metabolism<br/>Leukocytes, Mononuclear<br/>Mice<br/>Mice, Transgenic<br/>Oncostatin M<br/>Pancreas--immunology<br/>Peptides--physiology<br/>RNA, Messenger--analysis<br/>Sympathetic Nervous System<br/>Transgenes<br/>Vasoactive Intestinal Peptide--genetics<br/>Wound Healing--physiology