Costanzi-Strauss, E <br/><br/>
Restoration of growth arrest by p16INK4, p21WAF1, pRB, and p53 is dependent on the integrity of the endogenous cell-cycle control pathways in human glioblastoma cell lines.  [electronic resource] 

 -  Experimental cell research  Jan 1998 
 - 51-62 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0014-4827 
<br/><br/><label>Standard No.: </label>10.1006/excr.1997.3810  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Carrier Proteins--biosynthesis<br/>Cell Cycle--physiology<br/>Cell Cycle Proteins<br/>Cell Division<br/>Cyclin-Dependent Kinase Inhibitor p15<br/>Cyclin-Dependent Kinase Inhibitor p16<br/>Cyclin-Dependent Kinase Inhibitor p21<br/>Cyclins--biosynthesis<br/>Enzyme Inhibitors--metabolism<br/>Glioblastoma<br/>Humans<br/>Kinetics<br/>Recombinant Proteins--metabolism<br/>Retinoblastoma Protein--biosynthesis<br/>Retroviridae<br/>Transfection<br/>Tumor Cells, Cultured<br/>Tumor Suppressor Protein p53--biosynthesis<br/>Tumor Suppressor Proteins