Inhibition of protein tyrosine kinases attenuates increases in expression of transforming growth factor-beta isoforms and their receptors following arterial injury. [electronic resource]
- Arteriosclerosis, thrombosis, and vascular biology Nov 1997
- 2461-70 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1079-5642
10.1161/01.atv.17.11.2461 doi
Activin Receptors, Type I Angioplasty, Balloon--adverse effects Animals Antigens, CD--biosynthesis Carotid Arteries--metabolism Carotid Artery Injuries Cells, Cultured Enzyme Inhibitors--pharmacology Gene Expression Regulation--drug effects Genistein--pharmacology Integrin alphaV Integrin beta3 Isoflavones--pharmacology Male Muscle, Smooth, Vascular--drug effects Platelet Membrane Glycoproteins--biosynthesis Protein Serine-Threonine Kinases--biosynthesis Protein-Tyrosine Kinases--antagonists & inhibitors RNA, Messenger--biosynthesis Rats Rats, Sprague-Dawley Receptor, Transforming Growth Factor-beta Type I Receptor, Transforming Growth Factor-beta Type II Receptors, Growth Factor--biosynthesis Receptors, Transforming Growth Factor beta--biosynthesis Signal Transduction Transforming Growth Factor beta--biosynthesis Tunica Intima--injuries Wound Healing--physiology