Ward, M R

Inhibition of protein tyrosine kinases attenuates increases in expression of transforming growth factor-beta isoforms and their receptors following arterial injury. [electronic resource] - Arteriosclerosis, thrombosis, and vascular biology Nov 1997 - 2461-70 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1079-5642

10.1161/01.atv.17.11.2461 doi


Activin Receptors, Type I
Angioplasty, Balloon--adverse effects
Animals
Antigens, CD--biosynthesis
Carotid Arteries--metabolism
Carotid Artery Injuries
Cells, Cultured
Enzyme Inhibitors--pharmacology
Gene Expression Regulation--drug effects
Genistein--pharmacology
Integrin alphaV
Integrin beta3
Isoflavones--pharmacology
Male
Muscle, Smooth, Vascular--drug effects
Platelet Membrane Glycoproteins--biosynthesis
Protein Serine-Threonine Kinases--biosynthesis
Protein-Tyrosine Kinases--antagonists & inhibitors
RNA, Messenger--biosynthesis
Rats
Rats, Sprague-Dawley
Receptor, Transforming Growth Factor-beta Type I
Receptor, Transforming Growth Factor-beta Type II
Receptors, Growth Factor--biosynthesis
Receptors, Transforming Growth Factor beta--biosynthesis
Signal Transduction
Transforming Growth Factor beta--biosynthesis
Tunica Intima--injuries
Wound Healing--physiology