Tang, T <br/><br/>
A role for Mac-1 (CDIIb/CD18) in immune complex-stimulated neutrophil function in vivo: Mac-1 deficiency abrogates sustained Fcgamma receptor-dependent neutrophil adhesion and complement-dependent proteinuria in acute glomerulonephritis.  [electronic resource] 

 -  The Journal of experimental medicine  Dec 1997 
 - 1853-63 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0022-1007 
<br/><br/><label>Standard No.: </label>10.1084/jem.186.11.1853  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Actins--metabolism<br/>Acute Disease<br/>Animals<br/>Anti-Glomerular Basement Membrane Disease--complications<br/>Basement Membrane--immunology<br/>Capillary Permeability<br/>Cell Adhesion<br/>Complement C3b--deficiency<br/>Complement System Proteins--immunology<br/>Endothelium, Vascular--pathology<br/>Female<br/>Immune Complex Diseases--complications<br/>Intercellular Adhesion Molecule-1--genetics<br/>Isoantibodies--immunology<br/>Kidney Glomerulus--immunology<br/>Leukotriene B4--biosynthesis<br/>Macrophage-1 Antigen--genetics<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Models, Immunological<br/>Neutrophils--immunology<br/>Proteinuria--etiology<br/>Receptors, IgG--physiology