Yamamoto, K <br/><br/>
The kidneys of mice with autoimmune disease acquire a hypofibrinolytic/procoagulant state that correlates with the development of glomerulonephritis and tissue microthrombosis.  [electronic resource] 

 -  The American journal of pathology  Sep 1997 
 - 725-34 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0002-9440 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aging<br/>Animals<br/>Brain--metabolism<br/>Female<br/>Fibrin--metabolism<br/>In Situ Hybridization<br/>Kidney--drug effects<br/>Lipopolysaccharides--pharmacology<br/>Liver--metabolism<br/>Lung--metabolism<br/>Lupus Vulgaris--metabolism<br/>Mice<br/>Mice, Inbred Strains<br/>Myocardium--metabolism<br/>Plasminogen Activator Inhibitor 1--blood<br/>Polymerase Chain Reaction<br/>RNA, Messenger--metabolism<br/>Spleen--metabolism<br/>Thromboplastin--genetics<br/>Tissue Distribution<br/>Tissue Plasminogen Activator--genetics<br/>Urokinase-Type Plasminogen Activator--genetics