Hiyama, H <br/><br/>
Regulated ectopic expression of cyclin D1 induces transcriptional activation of the cdk inhibitor p21 gene without altering cell cycle progression.  [electronic resource] 

 -  Oncogene  May 1997 
 - 2533-42 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0950-9232 
<br/><br/><label>Standard No.: </label>10.1038/sj.onc.1201080  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>3T3 Cells<br/>Animals<br/>Base Sequence<br/>Blotting, Western<br/>Carrier Proteins<br/>Cell Cycle<br/>Cell Cycle Proteins--metabolism<br/>Cell Division--drug effects<br/>Cell Size--drug effects<br/>Cyclin D1<br/>Cyclin-Dependent Kinase 4<br/>Cyclin-Dependent Kinase Inhibitor p21<br/>Cyclin-Dependent Kinases--antagonists & inhibitors<br/>Cyclins--genetics<br/>DNA-Binding Proteins<br/>E2F Transcription Factors<br/>E2F1 Transcription Factor<br/>Enzyme Inhibitors--metabolism<br/>Fibroblasts--drug effects<br/>Gene Expression Regulation<br/>Glioma--metabolism<br/>Humans<br/>Immunohistochemistry<br/>Mice<br/>Models, Biological<br/>Oncogene Proteins--metabolism<br/>Proto-Oncogene Proteins<br/>Retinoblastoma-Binding Protein 1<br/>Tetracycline--pharmacology<br/>Time Factors<br/>Transcription Factor DP1<br/>Transcription Factors--metabolism<br/>Transcriptional Activation<br/>Tumor Suppressor Protein p53--metabolism