Sze, J Y <br/><br/>
VP16-activation of the C. elegans neural specification transcription factor UNC-86 suppresses mutations in downstream genes and causes defects in neural migration and axon outgrowth.  [electronic resource] 

 -  Development (Cambridge, England)  Mar 1997 
 - 1159-68 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0950-1991 
<br/><br/><label>Standard No.: </label>10.1242/dev.124.6.1159  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Axons--physiology<br/>Caenorhabditis elegans--genetics<br/>Caenorhabditis elegans Proteins<br/>Cell Differentiation<br/>Etoposide--pharmacology<br/>Gene Expression Regulation, Developmental<br/>Genes, Helminth<br/>Genetic Complementation Test<br/>Helminth Proteins--biosynthesis<br/>Herpes Simplex Virus Protein Vmw65--biosynthesis<br/>Homeodomain Proteins--metabolism<br/>LIM-Homeodomain Proteins<br/>Nervous System--cytology<br/>Nervous System Physiological Phenomena<br/>Neurons--cytology<br/>POU Domain Factors<br/>Recombinant Fusion Proteins--biosynthesis<br/>Regulatory Sequences, Nucleic Acid<br/>Transcription Factors--biosynthesis