Hart, K C <br/><br/>
Derivatives of activated H-ras lacking C-terminal lipid modifications retain transforming ability if targeted to the correct subcellular location.  [electronic resource] 

 -  Oncogene  Feb 1997 
 - 945-53 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0950-9232 
<br/><br/><label>Standard No.: </label>10.1038/sj.onc.1200908  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>3T3 Cells<br/>Animals<br/>Cell Compartmentation<br/>Cell Membrane--metabolism<br/>Fluorescent Antibody Technique, Indirect<br/>GTP Phosphohydrolases--metabolism<br/>Golgi Apparatus--metabolism<br/>Intracellular Membranes--metabolism<br/>Lipid Metabolism<br/>Membrane Proteins--chemistry<br/>Mice<br/>Myristates<br/>Protein Processing, Post-Translational<br/>Proto-Oncogene Proteins p21(ras)--metabolism<br/>Recombinant Fusion Proteins<br/>Structure-Activity Relationship