Wang, D Z <br/><br/>
Mutation in Sos1 dominantly enhances a weak allele of the EGFR, demonstrating a requirement for Sos1 in EGFR signaling and development.  [electronic resource] 

 -  Genes & development  Feb 1997 
 - 309-20 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0890-9369 
<br/><br/><label>Standard No.: </label>10.1101/gad.11.3.309  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alleles<br/>Animals<br/>Calcium-Calmodulin-Dependent Protein Kinases--metabolism<br/>Embryo, Mammalian--abnormalities<br/>Enhancer Elements, Genetic<br/>Enzyme Activation<br/>ErbB Receptors--genetics<br/>Female<br/>Fetal Death<br/>Fibroblasts<br/>Gene Deletion<br/>Gene Targeting<br/>Genes, Dominant<br/>Guanine Nucleotide Exchange Factors<br/>Heart Defects, Congenital--embryology<br/>Hematopoietic Stem Cells<br/>Heterozygote<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mutagenesis<br/>Phenotype<br/>Proteins--genetics<br/>Signal Transduction<br/>Stem Cells<br/>ras Guanine Nucleotide Exchange Factors