Kim, H A <br/><br/>
Nf1-deficient mouse Schwann cells are angiogenic and invasive and can be induced to hyperproliferate: reversion of some phenotypes by an inhibitor of farnesyl protein transferase.  [electronic resource] 

 -  Molecular and cellular biology  Feb 1997 
 - 862-72 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0270-7306 
<br/><br/><label>Standard No.: </label>10.1128/MCB.17.2.862  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alkyl and Aryl Transferases<br/>Animals<br/>Axons<br/>Biomarkers--analysis<br/>Blood<br/>Cell Division<br/>Cells, Cultured<br/>Colforsin--pharmacology<br/>Cyclic AMP-Dependent Protein Kinases--metabolism<br/>Enzyme Activation<br/>Enzyme Inhibitors--pharmacology<br/>Glia Maturation Factor<br/>Hyperplasia<br/>Mice<br/>Mice, Mutant Strains<br/>Mutation<br/>Myelin P0 Protein--analysis<br/>Neoplasm Invasiveness<br/>Neovascularization, Pathologic--physiopathology<br/>Nerve Growth Factors--pharmacology<br/>Nerve Tissue Proteins--pharmacology<br/>Neurofibromatosis 1--pathology<br/>Neurofibromin 1<br/>Oligopeptides--pharmacology<br/>Phenotype<br/>Protein Prenylation<br/>Proteins--physiology<br/>Schwann Cells--pathology<br/>Transferases--antagonists & inhibitors