Vasoactive intestinal peptide (VIP) and pituitary adenylate cyclase-activating polypeptide (PACAP) potentiate the glutamate-evoked release of arachidonic acid from mouse cortical neurons. Evidence for a cAMP-independent mechanism. [electronic resource]
- The Journal of biological chemistry Sep 1996
- 23705-10 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
0021-9258
10.1074/jbc.271.39.23705 doi
Animals Arachidonic Acid--metabolism Calcium--physiology Cells, Cultured Cerebral Cortex--metabolism Cyclic AMP--physiology Cyclic AMP-Dependent Protein Kinases--physiology GTP-Binding Protein alpha Subunits, Gs--physiology Glutamates--physiology Mice Neuropeptides--physiology Neurotransmitter Agents--physiology Pituitary Adenylate Cyclase-Activating Polypeptide Receptors, AMPA--physiology Receptors, N-Methyl-D-Aspartate--physiology Receptors, Pituitary Adenylate Cyclase-Activating Polypeptide Receptors, Pituitary Hormone--physiology Signal Transduction Type C Phospholipases--physiology Vasoactive Intestinal Peptide--physiology