van Oosterwijk, M F <br/><br/>
The sensitivity of Cockayne's syndrome cells to DNA-damaging agents is not due to defective transcription-coupled repair of active genes.  [electronic resource] 

 -  Molecular and cellular biology  Aug 1996 
 - 4436-44 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0270-7306 
<br/><br/><label>Standard No.: </label>10.1128/MCB.16.8.4436  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>2-Acetylaminofluorene<br/>Cell Survival--drug effects<br/>Cells, Cultured<br/>Cockayne Syndrome--genetics<br/>DNA Adducts--metabolism<br/>DNA Damage<br/>DNA Repair<br/>DNA Replication<br/>Gene Expression<br/>Humans<br/>RNA--biosynthesis<br/>Transcription, Genetic<br/>Ultraviolet Rays