Miller, R A <br/><br/>
Protease cleavage of iron-transferrin augments pyocyanin-mediated endothelial cell injury via promotion of hydroxyl radical formation.  [electronic resource] 

 -  Infection and immunity  Jan 1996 
 - 182-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0019-9567 
<br/><br/><label>Standard No.: </label>10.1128/iai.64.1.182-188.1996  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Drug Interactions<br/>Electron Spin Resonance Spectroscopy<br/>Endothelium, Vascular--drug effects<br/>Hydroxyl Radical--toxicity<br/>Iron--metabolism<br/>Lung--pathology<br/>Oxidation-Reduction<br/>Pancreatic Elastase--metabolism<br/>Pneumonia, Bacterial--pathology<br/>Pseudomonas aeruginosa--enzymology<br/>Pulmonary Artery--cytology<br/>Pyocyanine--metabolism<br/>Swine<br/>Toxicity Tests<br/>Transferrin--metabolism