Beg, A A <br/><br/>
Tumor necrosis factor and interleukin-1 lead to phosphorylation and loss of I kappa B alpha: a mechanism for NF-kappa B activation.  [electronic resource] 

 -  Molecular and cellular biology  Jun 1993 
 - 3301-10 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0270-7306 
<br/><br/><label>Standard No.: </label>10.1128/mcb.13.6.3301-3310.1993  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Antibodies, Monoclonal<br/>Antioxidants--pharmacology<br/>Blotting, Western<br/>Cell Nucleus--metabolism<br/>Cytoplasm--metabolism<br/>DNA-Binding Proteins--isolation & purification<br/>HeLa Cells<br/>Homeostasis<br/>Humans<br/>I-kappa B Proteins<br/>Interleukin-1--pharmacology<br/>Lipopolysaccharides--pharmacology<br/>Models, Biological<br/>NF-kappa B--isolation & purification<br/>Phosphates--metabolism<br/>Phosphorylation<br/>Phytohemagglutinins--pharmacology<br/>Tetradecanoylphorbol Acetate--pharmacology<br/>Tumor Cells, Cultured<br/>Tumor Necrosis Factor-alpha--pharmacology