Frauman, A G <br/><br/>
Nonimmune thyroid destruction results from transgenic overexpression of an allogeneic major histocompatibility complex class I protein.  [electronic resource] 

 -  Molecular and cellular biology  Mar 1993 
 - 1554-64 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article 
<br/><br/><label>ISSN: </label>0270-7306 
<br/><br/><label>Standard No.: </label>10.1128/mcb.13.3.1554-1564.1993  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Atrophy<br/>Autoimmune Diseases--etiology<br/>Crosses, Genetic<br/>Cytotoxicity, Immunologic<br/>DNA, Recombinant--genetics<br/>Fluorescent Antibody Technique<br/>Genes, MHC Class I--genetics<br/>H-2 Antigens--biosynthesis<br/>Heterozygote<br/>Homozygote<br/>Isoantigens--biosynthesis<br/>Mice<br/>Mice, Inbred Strains<br/>Mice, Transgenic--immunology<br/>Promoter Regions, Genetic--genetics<br/>Rats--genetics<br/>Recombinant Proteins--biosynthesis<br/>T-Lymphocytes, Cytotoxic<br/>Thyroglobulin--genetics<br/>Thyroid Gland--pathology