Wallace, W <br/><br/>
Amyloid precursor protein in the cerebral cortex is rapidly and persistently induced by loss of subcortical innervation.  [electronic resource] 

 -  Proceedings of the National Academy of Sciences of the United States of America  Sep 1993 
 - 8712-6 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0027-8424 
<br/><br/><label>Standard No.: </label>10.1073/pnas.90.18.8712  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>5,7-Dihydroxytryptamine--toxicity<br/>Acetylcholine--metabolism<br/>Actins--biosynthesis<br/>Afferent Pathways--drug effects<br/>Amyloid beta-Protein Precursor--biosynthesis<br/>Animals<br/>Cerebral Cortex--drug effects<br/>Choline O-Acetyltransferase--metabolism<br/>Diabetes Mellitus, Experimental--metabolism<br/>Functional Laterality<br/>Glucocorticoids--pharmacology<br/>Kinetics<br/>Lidocaine--pharmacology<br/>Male<br/>N-Methylaspartate--toxicity<br/>Norepinephrine--physiology<br/>Oxidopamine--toxicity<br/>Physostigmine--pharmacology<br/>Polyribosomes--metabolism<br/>RNA, Messenger--analysis<br/>Raphe Nuclei--drug effects<br/>Rats<br/>Rats, Sprague-Dawley<br/>Substantia Innominata--drug effects<br/>Time Factors