Cannon, S C <br/><br/>
Loss of Na+ channel inactivation by anemone toxin (ATX II) mimics the myotonic state in hyperkalaemic periodic paralysis.  [electronic resource] 

 -  The Journal of physiology  Jul 1993 
 - 501-20 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-3751 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Action Potentials--drug effects<br/>Animals<br/>Cnidarian Venoms--pharmacology<br/>Disease Models, Animal<br/>Female<br/>Humans<br/>Hyperkalemia--etiology<br/>In Vitro Techniques<br/>Isometric Contraction--drug effects<br/>Muscles--drug effects<br/>Mutation<br/>Myotonia--etiology<br/>Paralyses, Familial Periodic--etiology<br/>Rats<br/>Sea Anemones<br/>Sodium Channel Blockers<br/>Sodium Channels--genetics