Ayala, A <br/><br/>
Mechanism of enhanced susceptibility to sepsis following hemorrhage. Interleukin-10 suppression of T-cell response is mediated by eicosanoid-induced interleukin-4 release.  [electronic resource] 

 -  Archives of surgery (Chicago, Ill. : 1960)  Nov 1994 
 - 1172-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0004-0010 
<br/><br/><label>Standard No.: </label>10.1001/archsurg.1994.01420350070009  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Disease Susceptibility<br/>Eicosanoids--physiology<br/>Hemorrhage--immunology<br/>Interleukin-10--immunology<br/>Interleukin-4--metabolism<br/>Macrophages--immunology<br/>Male<br/>Mice<br/>Mice, Inbred C3H<br/>Sepsis--immunology<br/>Spleen--immunology<br/>T-Lymphocytes--immunology