Aggarwal, B B <br/><br/>
pp60v-src kinase overexpression leads to cellular resistance to the antiproliferative effects of tumor necrosis factor.  [electronic resource] 

 -  FEBS letters  May 1994 
 - 219-24 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0014-5793 
<br/><br/><label>Standard No.: </label>10.1016/0014-5793(94)00441-2  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>3T3 Cells<br/>Animals<br/>Cell Division--drug effects<br/>Down-Regulation<br/>Drug Resistance--physiology<br/>ErbB Receptors--biosynthesis<br/>Glutathione--metabolism<br/>Humans<br/>Interferon-gamma--pharmacology<br/>Kinetics<br/>Mice<br/>Oncogene Protein pp60(v-src)--biosynthesis<br/>Proto-Oncogene Proteins--biosynthesis<br/>Receptor Protein-Tyrosine Kinases--biosynthesis<br/>Receptor, ErbB-2<br/>Receptors, Tumor Necrosis Factor--biosynthesis<br/>Recombinant Proteins<br/>Time Factors<br/>Transfection<br/>Tumor Necrosis Factor-alpha--toxicity