Ito, M <br/><br/>
Nephrosis augments contractile response to adrenoceptor agonists by the decrease in release of endothelium-derived relaxing factor from the endothelial cells.  [electronic resource] 

 -  The Journal of pharmacology and experimental therapeutics  May 1994 
 - 589-95 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>0022-3565 
<br/><br/><label>Subjects--Topical Terms: </label><br/>15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid<br/>Adrenergic alpha-Agonists--pharmacology<br/>Animals<br/>Azepines--pharmacology<br/>Cyclic GMP--metabolism<br/>Endothelium, Vascular--drug effects<br/>Male<br/>Muscle Contraction--drug effects<br/>Nephrosis--metabolism<br/>Nitric Oxide--metabolism<br/>Norepinephrine--pharmacology<br/>Potassium Chloride--pharmacology<br/>Prostaglandin Endoperoxides, Synthetic--pharmacology<br/>Rats<br/>Rats, Sprague-Dawley<br/>Thromboxane A2--analogs & derivatives