Diminished responsiveness of senescent normal human fibroblasts to TNF-dependent proliferation and interleukin production is not due to its effect on the receptors or on the activation of a nuclear factor NF-kappa B. [electronic resource]
- Experimental cell research May 1995
- 381-8 p. digital
Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
0014-4827
10.1006/excr.1995.1169 doi
Base Sequence Binding Sites Cell Division--drug effects Cell Nucleus--drug effects Cells, Cultured Cellular Senescence Cytokines--pharmacology Fibroblast Growth Factor 2--pharmacology Fibroblasts--cytology HIV Long Terminal Repeat Humans Infant, Newborn Interleukin-1--pharmacology Interleukin-6--biosynthesis Interleukin-8--biosynthesis Interleukins--biosynthesis Kinetics Male Molecular Sequence Data NF-kappa B--metabolism Oligodeoxyribonucleotides Receptors, Tumor Necrosis Factor--metabolism Skin--cytology Tumor Necrosis Factor-alpha--metabolism