Wang, Z Q <br/><br/>
Mice lacking ADPRT and poly(ADP-ribosyl)ation develop normally but are susceptible to skin disease.  [electronic resource] 

 -  Genes & development  Mar 1995 
 - 509-20 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0890-9369 
<br/><br/><label>Standard No.: </label>10.1101/gad.9.5.509  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Base Sequence<br/>Cell Division<br/>Chimera<br/>DNA Repair--physiology<br/>Disease Susceptibility<br/>Epidermis--pathology<br/>Fetus--enzymology<br/>Fibroblasts--physiology<br/>Gamma Rays<br/>Germ-Line Mutation<br/>Hyperplasia<br/>Mice<br/>Mice, Knockout<br/>Molecular Sequence Data<br/>Poly Adenosine Diphosphate Ribose--analysis<br/>Poly(ADP-ribose) Polymerases--analysis<br/>Protein Processing, Post-Translational--physiology<br/>RNA, Messenger--analysis<br/>Skin Diseases--etiology<br/>Thymus Gland--cytology