Sacchi, N <br/><br/>
AML1 fusion transcripts in t(3;21) positive leukemia: evidence of molecular heterogeneity and usage of splicing sites frequently involved in the generation of normal AML1 transcripts.  [electronic resource] 

 -  Genes, chromosomes & cancer  Dec 1994 
 - 226-36 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1045-2257 
<br/><br/><label>Standard No.: </label>10.1002/gcc.2870110405  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amino Acid Sequence<br/>Base Sequence<br/>Blast Crisis--genetics<br/>Chromosome Banding<br/>Chromosomes, Human, Pair 21<br/>Chromosomes, Human, Pair 3<br/>Cloning, Molecular<br/>Core Binding Factor Alpha 2 Subunit<br/>DNA Probes<br/>DNA-Binding Proteins<br/>Exons<br/>Humans<br/>In Situ Hybridization, Fluorescence<br/>Karyotyping<br/>Leukemia, Myelogenous, Chronic, BCR-ABL Positive--genetics<br/>Leukemia, Myeloid, Acute--genetics<br/>Molecular Sequence Data<br/>Neoplasm Proteins--chemistry<br/>Proto-Oncogene Proteins<br/>RNA Splicing<br/>RNA, Messenger--genetics<br/>Recombinant Fusion Proteins--chemistry<br/>Transcription Factors<br/>Translocation, Genetic