DeGregori, J <br/><br/>
E2F-1 accumulation bypasses a G1 arrest resulting from the inhibition of G1 cyclin-dependent kinase activity.  [electronic resource] 

 -  Genes & development  Dec 1995 
 - 2873-87 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0890-9369 
<br/><br/><label>Standard No.: </label>10.1101/gad.9.23.2873  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Blotting, Western<br/>CDC2-CDC28 Kinases<br/>Carrier Proteins--genetics<br/>Cell Cycle Proteins<br/>Cyclin-Dependent Kinase 2<br/>Cyclin-Dependent Kinase Inhibitor p16<br/>Cyclin-Dependent Kinase Inhibitor p27<br/>Cyclin-Dependent Kinases--antagonists & inhibitors<br/>DNA--radiation effects<br/>DNA-Binding Proteins<br/>E2F Transcription Factors<br/>E2F1 Transcription Factor<br/>G1 Phase--radiation effects<br/>G2 Phase<br/>Gamma Rays<br/>Gene Expression Regulation<br/>Microtubule-Associated Proteins--genetics<br/>Mitosis<br/>Plasmids<br/>Promoter Regions, Genetic<br/>Protein Serine-Threonine Kinases--genetics<br/>Retinoblastoma Protein--biosynthesis<br/>Retinoblastoma-Binding Protein 1<br/>S Phase<br/>Transcription Factors--genetics<br/>Tumor Suppressor Proteins