Honegger, A M <br/><br/>
Point mutation at the ATP binding site of EGF receptor abolishes protein-tyrosine kinase activity and alters cellular routing.  [electronic resource] 

 -  Cell  Oct 1987 
 - 199-209 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>0092-8674 
<br/><br/><label>Standard No.: </label>10.1016/0092-8674(87)90147-4  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenosine Triphosphate--metabolism<br/>Binding Sites<br/>Cell Line<br/>Epidermal Growth Factor--pharmacokinetics<br/>ErbB Receptors--drug effects<br/>Humans<br/>Mutation<br/>Protein-Tyrosine Kinases--metabolism