Huang, Chun-Ta <br/><br/>
Glycemic control with insulin attenuates sepsis-associated encephalopathy by inhibiting glial activation via the suppression of the nuclear factor kappa B and mitogen-activated protein kinase signaling pathways in septic rats.  [electronic resource] 

 -  Brain research  07 2020 
 - 146822 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1872-6240 
<br/><br/><label>Standard No.: </label>10.1016/j.brainres.2020.146822  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Blood Glucose--metabolism<br/>Brain Diseases--drug therapy<br/>Disease Models, Animal<br/>Glycemic Control--methods<br/>Hyperglycemia--metabolism<br/>Insulin--metabolism<br/>MAP Kinase Signaling System<br/>Male<br/>Mitogen-Activated Protein Kinases--metabolism<br/>NF-kappa B--metabolism<br/>Neuroglia--metabolism<br/>Rats<br/>Rats, Sprague-Dawley<br/>Sepsis--complications<br/>Sepsis-Associated Encephalopathy--drug therapy<br/>Signal Transduction--physiology<br/>Tumor Necrosis Factor-alpha--metabolism